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dusp4 mouse monoclonal antibody  (Millipore)

 
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    Millipore dusp4 mouse monoclonal antibody
    qPCR validation of new genes that are differentially expressed in Shp−/− mice, human liver steatosis, fibrosis, NASH, and alcohol and hepatitis C cirrhosis. A: from left to right, peptidoglycan recognition protein 2 (PGLYRP2), dual specific phosphatase-4 <t>(DUSP4),</t> and tetraspanin 4 (TSPAN4). B: from left to right, thrombospondin 1 (THBS1), SPARC-related modular calcium binding protein-2 (SMOC2), and SHP. Expression levels of each gene for each sample were normalized to the hypoxanthine phosphoribosyltransferase 1 expression level of that sample as an internal control. ¥P <0.001, ‡P <0.01, and *P <0.05. The no. of specimens in each group is as follows: HCV cirrhosis (n = 39), alcohol cirrhosis (n = 28), NASH cirrhosis (n = 13), fibrosis (n = 7), steatosis (n = 15), and control (n = 24). C: immunohistochemistry analysis of DUSP4 protein in human alcohol and hepatitis C cirrhosis compared with the normal liver. Two representative results from each group are shown. N, normal; AC, alcohol cirrhosis; HC, HCV cirrhosis. Top: DUSP4 protein expression. Bottom: DUSP4 overlay with DAPI staining. Magnification ×20.
    Dusp4 Mouse Monoclonal Antibody, supplied by Millipore, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/dusp4 mouse monoclonal antibody/product/Millipore
    Average 90 stars, based on 1 article reviews
    dusp4 mouse monoclonal antibody - by Bioz Stars, 2026-02
    90/100 stars

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    1) Product Images from "Genome-wide transcriptome analysis identifies novel gene signatures implicated in human chronic liver disease"

    Article Title: Genome-wide transcriptome analysis identifies novel gene signatures implicated in human chronic liver disease

    Journal: American Journal of Physiology - Gastrointestinal and Liver Physiology

    doi: 10.1152/ajpgi.00077.2013

    qPCR validation of new genes that are differentially expressed in Shp−/− mice, human liver steatosis, fibrosis, NASH, and alcohol and hepatitis C cirrhosis. A: from left to right, peptidoglycan recognition protein 2 (PGLYRP2), dual specific phosphatase-4 (DUSP4), and tetraspanin 4 (TSPAN4). B: from left to right, thrombospondin 1 (THBS1), SPARC-related modular calcium binding protein-2 (SMOC2), and SHP. Expression levels of each gene for each sample were normalized to the hypoxanthine phosphoribosyltransferase 1 expression level of that sample as an internal control. ¥P <0.001, ‡P <0.01, and *P <0.05. The no. of specimens in each group is as follows: HCV cirrhosis (n = 39), alcohol cirrhosis (n = 28), NASH cirrhosis (n = 13), fibrosis (n = 7), steatosis (n = 15), and control (n = 24). C: immunohistochemistry analysis of DUSP4 protein in human alcohol and hepatitis C cirrhosis compared with the normal liver. Two representative results from each group are shown. N, normal; AC, alcohol cirrhosis; HC, HCV cirrhosis. Top: DUSP4 protein expression. Bottom: DUSP4 overlay with DAPI staining. Magnification ×20.
    Figure Legend Snippet: qPCR validation of new genes that are differentially expressed in Shp−/− mice, human liver steatosis, fibrosis, NASH, and alcohol and hepatitis C cirrhosis. A: from left to right, peptidoglycan recognition protein 2 (PGLYRP2), dual specific phosphatase-4 (DUSP4), and tetraspanin 4 (TSPAN4). B: from left to right, thrombospondin 1 (THBS1), SPARC-related modular calcium binding protein-2 (SMOC2), and SHP. Expression levels of each gene for each sample were normalized to the hypoxanthine phosphoribosyltransferase 1 expression level of that sample as an internal control. ¥P <0.001, ‡P <0.01, and *P <0.05. The no. of specimens in each group is as follows: HCV cirrhosis (n = 39), alcohol cirrhosis (n = 28), NASH cirrhosis (n = 13), fibrosis (n = 7), steatosis (n = 15), and control (n = 24). C: immunohistochemistry analysis of DUSP4 protein in human alcohol and hepatitis C cirrhosis compared with the normal liver. Two representative results from each group are shown. N, normal; AC, alcohol cirrhosis; HC, HCV cirrhosis. Top: DUSP4 protein expression. Bottom: DUSP4 overlay with DAPI staining. Magnification ×20.

    Techniques Used: Binding Assay, Expressing, Immunohistochemistry, Staining



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    dusp4 mouse monoclonal antibody  (Millipore)
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    qPCR validation of new genes that are differentially expressed in Shp−/− mice, human liver steatosis, fibrosis, NASH, and alcohol and hepatitis C cirrhosis. A: from left to right, peptidoglycan recognition protein 2 (PGLYRP2), dual specific phosphatase-4 <t>(DUSP4),</t> and tetraspanin 4 (TSPAN4). B: from left to right, thrombospondin 1 (THBS1), SPARC-related modular calcium binding protein-2 (SMOC2), and SHP. Expression levels of each gene for each sample were normalized to the hypoxanthine phosphoribosyltransferase 1 expression level of that sample as an internal control. ¥P <0.001, ‡P <0.01, and *P <0.05. The no. of specimens in each group is as follows: HCV cirrhosis (n = 39), alcohol cirrhosis (n = 28), NASH cirrhosis (n = 13), fibrosis (n = 7), steatosis (n = 15), and control (n = 24). C: immunohistochemistry analysis of DUSP4 protein in human alcohol and hepatitis C cirrhosis compared with the normal liver. Two representative results from each group are shown. N, normal; AC, alcohol cirrhosis; HC, HCV cirrhosis. Top: DUSP4 protein expression. Bottom: DUSP4 overlay with DAPI staining. Magnification ×20.
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    qPCR validation of new genes that are differentially expressed in Shp−/− mice, human liver steatosis, fibrosis, NASH, and alcohol and hepatitis C cirrhosis. A: from left to right, peptidoglycan recognition protein 2 (PGLYRP2), dual specific phosphatase-4 <t>(DUSP4),</t> and tetraspanin 4 (TSPAN4). B: from left to right, thrombospondin 1 (THBS1), SPARC-related modular calcium binding protein-2 (SMOC2), and SHP. Expression levels of each gene for each sample were normalized to the hypoxanthine phosphoribosyltransferase 1 expression level of that sample as an internal control. ¥P <0.001, ‡P <0.01, and *P <0.05. The no. of specimens in each group is as follows: HCV cirrhosis (n = 39), alcohol cirrhosis (n = 28), NASH cirrhosis (n = 13), fibrosis (n = 7), steatosis (n = 15), and control (n = 24). C: immunohistochemistry analysis of DUSP4 protein in human alcohol and hepatitis C cirrhosis compared with the normal liver. Two representative results from each group are shown. N, normal; AC, alcohol cirrhosis; HC, HCV cirrhosis. Top: DUSP4 protein expression. Bottom: DUSP4 overlay with DAPI staining. Magnification ×20.
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    Image Search Results


    qPCR validation of new genes that are differentially expressed in Shp−/− mice, human liver steatosis, fibrosis, NASH, and alcohol and hepatitis C cirrhosis. A: from left to right, peptidoglycan recognition protein 2 (PGLYRP2), dual specific phosphatase-4 (DUSP4), and tetraspanin 4 (TSPAN4). B: from left to right, thrombospondin 1 (THBS1), SPARC-related modular calcium binding protein-2 (SMOC2), and SHP. Expression levels of each gene for each sample were normalized to the hypoxanthine phosphoribosyltransferase 1 expression level of that sample as an internal control. ¥P <0.001, ‡P <0.01, and *P <0.05. The no. of specimens in each group is as follows: HCV cirrhosis (n = 39), alcohol cirrhosis (n = 28), NASH cirrhosis (n = 13), fibrosis (n = 7), steatosis (n = 15), and control (n = 24). C: immunohistochemistry analysis of DUSP4 protein in human alcohol and hepatitis C cirrhosis compared with the normal liver. Two representative results from each group are shown. N, normal; AC, alcohol cirrhosis; HC, HCV cirrhosis. Top: DUSP4 protein expression. Bottom: DUSP4 overlay with DAPI staining. Magnification ×20.

    Journal: American Journal of Physiology - Gastrointestinal and Liver Physiology

    Article Title: Genome-wide transcriptome analysis identifies novel gene signatures implicated in human chronic liver disease

    doi: 10.1152/ajpgi.00077.2013

    Figure Lengend Snippet: qPCR validation of new genes that are differentially expressed in Shp−/− mice, human liver steatosis, fibrosis, NASH, and alcohol and hepatitis C cirrhosis. A: from left to right, peptidoglycan recognition protein 2 (PGLYRP2), dual specific phosphatase-4 (DUSP4), and tetraspanin 4 (TSPAN4). B: from left to right, thrombospondin 1 (THBS1), SPARC-related modular calcium binding protein-2 (SMOC2), and SHP. Expression levels of each gene for each sample were normalized to the hypoxanthine phosphoribosyltransferase 1 expression level of that sample as an internal control. ¥P <0.001, ‡P <0.01, and *P <0.05. The no. of specimens in each group is as follows: HCV cirrhosis (n = 39), alcohol cirrhosis (n = 28), NASH cirrhosis (n = 13), fibrosis (n = 7), steatosis (n = 15), and control (n = 24). C: immunohistochemistry analysis of DUSP4 protein in human alcohol and hepatitis C cirrhosis compared with the normal liver. Two representative results from each group are shown. N, normal; AC, alcohol cirrhosis; HC, HCV cirrhosis. Top: DUSP4 protein expression. Bottom: DUSP4 overlay with DAPI staining. Magnification ×20.

    Article Snippet: Detection of dual specific phosphatase-4 (DUSP4) protein levels was carried out using a DUSP4 mouse monoclonal antibody (SAB1403748-100UG; Sigma-Aldrich).

    Techniques: Binding Assay, Expressing, Immunohistochemistry, Staining